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Prolonged hyperglycemia accelerates this glycation process, resulting in the accumulation of AGEs both endogenously through metabolic pathways and exogenously from dietary sources
Our images showed that GLP-1R-positive cells were mainly double labelled with Iba-1 and GFAP but did not colocalize with NeuN (Fig
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Rodents, contrarily to humans, express GPR119 also in some regions of the brain (316)
