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More extensive analysis of signaling and regulatory pathways that included recruitment of arrestins revealed additional tiers of biased signaling with both exendin and oxyntomdulin exhibiting stronger arrestin recruitment relative to GLP-1 736 amide , whereas GLP-1 136 did not recruit arrestins (Wootten et al., 2013b) (Fig

doi: 10.1111/dom.14944

November 21, 2022

4,6 A key issue in the physiology of type 2 diabetes (T2DM) is the reduced incretin effect, which mainly results from decreased activity of the primary incretin hormones, GLP-1 (glucagon-like peptide 1) and GIP (glucose-dependent insulinotropic peptide)

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