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This C-terminal metabolite of GLP-1 has shown cytoprotective effects in isolated mouse hepatocytes and cells exposed to oxidative stress injury, as indicated by trafficking to the mitochondria and the limiting of stress-induced apoptosis through inhibition of mitochondrial depolarization, cytochrome c release, and caspase activation (11, 42)

In a post-hoc analysis of the REWIND trial, the incidence of de novo atrial arrhythmias in patients with T2D over 5 years was similar in dulaglutide and placebo groups (5.6% and 5.3%, respectively

Dungan KM, Povedano ST, Forst T, Gonzalez JG, Atisso C, Sealls W, et al

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The reduction was significantly bigger in the two dulaglutide study arms than with exenatide: 1.51% compared with baseline in the dulaglutide 1.5-mg arm, 1.3% with dulaglutide 0.75 mg, 0.99% with exenatide, and 0.46% with placebo

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