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Beta-cell function progressively declines, often necessitating treatment intensification over time to maintain glycemic control
The conventional understanding of Alzheimer's disease frames it primarily as a problem of protein accumulation: amyloid plaques building up between neurons, tau tangles forming within them, and the resulting structural damage driving cognitive decline
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Heisler LK, Cowley MA, Kishi T, Tecott LH, Fan W, Low MJ, et al
GLP-1 therapies alter the way food moves in the gut and its fermentation patterns
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