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In addition, this review discusses the multilevel pathophysiological mechanisms involved in acute pancreatitis, including aberrant enzymatic activation, calcium overload, impaired autophagy, inflammatory responses, and various forms of pancreatic acinar cell death
Bridging the gap between guidelines and practice will require systemic interventions, including physician training, patient-centered care models, and devising research strategies such as national HF registries to understand and tailor therapeutic approaches from an Indian perspective.
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