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The new 5-HT hypothesis of depression: cell-mediated immune activation induces indoleamine 2,3-dioxygenase, which leads to lower plasma tryptophan and an increased synthesis of detrimental tryptophan catabolites (TRYCATs), both of which contribute to the onset of depression

This slower escalation compared to other medications reflects the triple mechanism requiring more gradual receptor adaptation

The analysis relied on Mendelian randomization assumptions, used only a small number of GLP1R-region instruments, and could not completely exclude effects from neighboring genes or other biological pathways

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