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Through HDAC6 inhibition, the pathway promotes the removal of damaged cellular components, reducing oxidative stress and enhancing cellular repair mechanisms in retinal cells ( 2.2.3 Beyond the eye In renal tubular cells ( + -dependent deacetylase SIRT1, reducing mitochondrial dysfunction and oxidative stress by decreasing ROS production, thereby preventing apoptosis induced by lipotoxicity

Perreault, L., et al

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However, the demand appears to be fast-tracking promising new research

Lower levels of lipids (except high-density lipoprotein, also called good cholesterol), which indicates a lower risk of heart disease and stroke

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