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107 GLP-1s ability to mediate the incretin effect is its most studied and significant physiological effect due to the use of GLP-1 analogues (exenatide and liraglutide, lixisenatide, dulaglutide, albiglutide and semaglutide) in T2D treatment

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On the other hand, phosphorylated tau protein and A disturb autophagy, which is a major event in AD pathogenesis (77)

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