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Liraglutide restored proper autophagic flux by alleviating the accumulation of P62 (an autophagy dysfunction marker) in the lacrimal gland, reduced oxidative damage by enhancing clearance of ROS and promoting cellular repair mechanisms, and potentially addressed dry eye syndrome (Sun et al., 2025)

a full label is still needed to independently confirm it

While use of the GLP-1 medications has skyrocketed, evidence on their long-term impact is still being assessed

Implications of cardiovascular outcome trials with injectable antidiabetic agents

(2003) suggested that the cleft may enable large peptide substrates to access the active site, whereas the narrower tunnel formed by the -propeller could serve as a conduit for exit of dipeptide cleavage products