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In in vitro studies of the effects of GLP-1 on pancreatic cells, GLP-1-treated human islets showed increased level of bcl-2 protein and decreased level of caspase-3 protein54)
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GLP-1R-mediated recruitment of arrestins also leads to transactivation of epidermal growth factor receptor, activation of phosphatidylinositol 3-kinase and downstream nuclear translocation of protein kinase C to promote beta cell proliferation, insulin gene expression, and insulin synthesis and secretion [27,36]
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