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S7A-E) not only appeared more crowded in groups E2 and E3, but also exhibited a more typical mesenchymal cell morphology, with spindle-shaped features (Fig

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The addition of omic layers refines both mechanistic insight and predictive accuracy: transcriptomic/inflammatory axes involving GLP-1R/GIPR signaling and IL-6 have been implicated in inter-individual incretin biology and GLP-1RA response heterogeneity, and metabolomic/lipidomic profilesparticularly elevated branched-chain amino acids and distinct triacylglycerol speciesprovide orthogonal markers linked to insulin resistance and cardiovascular risk [98,99]

2,5,8-10,12,13 As research advances, these insights highlight the potential of nutrition science to inform future GLP-1-targeted therapies and sustainable strategies for metabolic health