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10.1080/0886022X.2024.2441398 Summary Keywords gout, hyperuricemia, drug delivery systems, therapeutic, prospect Citation Peng Z, Meng F, Deng Q, Huang Y, Mao D, Long Y, Yan W, Peng J, Wang X and Liu N (2025) Advances in drug delivery systems for the management of gout and hyperuricemia

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Regulatory status (US) Not FDA-approved Category 2 (2023) PCAC July 2026 Compound Melatonin What it is Hormone supplement Mechanism (in plain English) Shifts sleep timing via the circadian system

The term immunosuppressive polynucleotide as used herein, represents an immunomodulating polynucleotide capable of antagonizing an immune response, as determined by e.g., a reduction in the activation or lack of activation of NFB or lack of increase in the levels of cell surface marker(s) of activation of function or a reduction or lack of increase in the secretion of at least one inflammatory cytokine or at least one type I interferon in an immune cell (e.g., antigen-presenting cell) to which an immunosuppressive polynucleotide was delivered (e.g., in comparison to another immune cell (e.g., antigen-presenting cell) to which an immunosuppressive polynucleotide was not delivered) or in an immune cell that interacts with an immune cell (e.g., antigen-presenting cell) to which an immunomodulating polynucleotide was delivered (including direct cell-to-cell interactions as well as indirect stimulation, e.g., from one or more cytokines secreted by the cell to which an immunomodulating polynucleotide was delivered)