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These findings demonstrate that tissue eosinophils undergo dynamic transcriptional reprogramming, acquiring context-specific phenotypes that reflect their roles in inflammation and tissue remodeling

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[DOI] [PubMed] [Google Scholar] 19.Chopko T.C., Lindsley C.W

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doi: 10.1001/archgenpsychiatry.2009.81 10 AlduhishyM

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Furthermore, NCOA4 knockdown in BEAS-2B reduced CSE-induced lipid peroxidation (C11BODIPY staining) and cell death (LDH and MTT assay), indicating that ferritin degradation via NCOA4-mediated ferritinophagy is responsible for ferroptosis in bronchial cell death during CS exposure (Fig

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Amber12EHT is an all-atom force field developed to calculate conformations of proteins, nucleic acids, and small molecules in [55]

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