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Powthong P, Suntornthiticharoen P

Since ROS exert positive and negative effects on cell proliferation and survival, intracellular ROS levels are regulated by multiple factors 25,29 , among which GSH appears to play a major role

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Furthermore, one of the haplotype blocks generated of the GSR gene involving 2 SNPs revealed that these variations may be responsible for obstructive heart defects ((rs2978663) OHDs) and hereditary anaemia (rs8190955) with different population frequencies

and (2) suppressing immune responses by activating NF-B, TNF, IL-10, IFN-, and GM-CSF, thereby increasing the susceptibility to infections (116, 117)