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The question of whether cagrilintide and semaglutide for fatty liver disease represents a meaningful research direction sits at the intersection of two of the most active fields in metabolic medicine: incretin-based weight loss and the treatment of metabolic dysfunctionassociated steatotic liver disease (MASLD)
About this article Cite this article Sibarov, D.A., Poguzhelskaya, E.E
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Subsequent research revealed multiple mechanisms behind its antinociceptive effect, including activation of nitric oxide (NO)-dependent cGMP pathways [209], CB2 cannabinoid receptor activation [210], and TRPA1 channel desensitization [211]
