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Increases in TMAO levels during CVD/T2D progression (beyond the observed wide natural interindividual variability in TMAO levels) could be the result of disease-related dysbiosis
The decrease in 18:1n9 was particularly noteworthy, given the high abundance of that FA (Fig
L-Carnitine Tartrate
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This enables the liver to buffer blood glucose and provide alternative fuel (ketone bodies) for highly oxidative tissues during food deprivation
