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In vivo studies in mice with acute hyperuricemia have shown that 47 and 48 can significantly decrease serum uric acid levels at a dosage of 5 mg.kg 1

A prospective observational study revealed that treatment with AST-120 for 24 weeks in CKD patients significantly decreased serum IS levels, and concurrently increased GSH to GSSG ratios, suggesting that AST-120 potentially has a direct effect on relieving oxidative stress in these patients [78]

40 This molecular mechanism is closely related to the role of SAM participating in methylation reactions to provide methyl groups, entering the transsulfuration pathway to metabolize and synthesize GSH, and participating in the one-carbon cycle

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For example, several kinases, such as MK2, TBK1, TAK1, and IKK/, prevent RIPK1 kinase activation and the subsequent cell death and inflammation by phosphorylating RIPK1 15,21,22,23