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As mentioned before, a critical limitation of these strategies lies in the degeneracy of dominant nociceptor subtypes that may dynamically shift following nerve injury or over time, diminishing the efficacy of highly selective monotherapies [361, 362]

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CBD promotes oxaliplatin-mediated autophagy via NOS 3 -mediated mitochondrial dysfunction, implying that NOS 3 is a viable therapeutic target for overcoming oxaliplatin resistance and that CBD could be a novel treatment option for CRC (Jeong et al

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USP9X prevents AGEs-induced upregulation of FN and TGF-1 through activating Nrf2-ARE pathway in rat glomerular mesangial cells