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Acute inhibition of the TCA cycle (TCAi) in mouse kidney epithelial cells results in increased glutathione synthesis and impaired proline and aspartate synthesis (Figure 2)

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You just have to get it compounded for you in a wet compounding pharmacy (usually privately owned, not corporate)

Two downstream consequences are mechanistically central: nicotinamide is preserved for re-entry into the NAD+ salvage pathway (raising intracellular NAD+ availability), and SAM is preserved for other methylation reactions

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