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In genetically engineered mouse models (GEMMs), constitutive deletion of Egfr , specifically in myeloid cells before tumor initiation, led to reduced tumor burden in CRC and HCC, whereas EGFR deletion in tumor cells (intestinal epithelial cells and hepatocytes) did not affect (CRC) or even enhanced (HCC) tumor growth [8, 9]

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Decreased activity of GPX4 prevents the metabolism of lipid peroxides via GPX4-catalyzed glutathione (GSH) reduction reactions, resulting in the oxidation of divalent iron ions and the generation of reactive oxygen species (ROS) in lipids (19)