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This residual risk is likely attributable at least in part to inflammation

The main difference is how many hormone pathways each drug targets

and (2) suppressing immune responses by activating NF-B, TNF, IL-10, IFN-, and GM-CSF, thereby increasing the susceptibility to infections (116, 117)

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The BiTE (bispecific T-cell engager) platform: Development and future potential of a targeted immuno-oncology therapy across tumor types