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this article addresses the cofactor at the GR end
Vet Sci 7:71
The 3A mutation in GAP-F5 protein effectively blocks the increase in G2019S LRRK2-mediated phosphorylation in response to increasing amounts of GAP-F5 protein substrate, compared to the concentration-dependent increase in WT GAP-F5 phosphorylation, by in vitro radioactive kinase assay ( Fig
B12 is found in many animal products and Vegans are at the highest risk of deficiency
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