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124 As an acetylation-defective mutant, p533KR cannot induce apoptosis, cell cycle arrest, or senescence, but it can regulate the expression of SLC7A11 and induce ferroptosis mediated by reactive oxygen species (ROS)

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All mice were sacrificed at the age of 12 weeks except for mice in the post-treatment group, which were sacrificed at 16 weeks of age

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While the functional consequence of this postranslational modification is unknown, these modifications should lead to changes in protein conformation and function