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This immunoevasive phenotype is mediated in part through diminished dendritic cell and T cell responses, a finding that was also recognized in human lung adenocarcinoma patient samples [60]

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Recent studies have demonstrated that adipose tissue contributes to the level of NAA in the blood and that this contribution is controlled by the activity of aspartoacylase

Validating these findings, we show that deletion of the mitochondrial pyruvate carrier, which is known to decrease TCA cycle activity and protect the liver from diet-induced injury, also diminished NADPH, elevated glutathione oxidation, and alleviated ER stress

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