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Insulin resistance drives hyperandrogenism, anovulation, and impaired endometrial function
Recently, the presence of nuclear-encoded transcription factors (MOF, AP1, CEBPB, MEF2D), and the contribution of nuclear spliceosome complex to mtDNA-splicing provided strong evidence of control of mitochondrial replication/transcription demonstrating evolutionary adaptation of the mammalian mtDNA in the nucleoid [75]
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SMD, standardized mean difference
98 IovineJ
