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About this article Cite this article Elbanna, A.H., El-Dessouki, A.M., Mageed, S.S.A

The core argument is that estrogen acts as an endogenous, multi-target inhibitor of ferroptosis, conferring robust anti-ferroptotic capabilities to vascular cells by activating the Nrf2 central antioxidant system, optimizing cellular iron homeostasis (e.g., upregulating FPN1 and inhibiting ferritinophagy), and exerting direct antioxidant effects

46 , 47 Conversely, low levels of CRT2 have been suggested to be associated with increased platinum accumulation throughout the cell and higher sensitivity to platinum

After HNO 3 pretreatment, gel phase DPPC liposomes were immobilized onto the SiO 2 sensor surface at 25 C

Early prediction of developing type 2 diabetes by plasma acylcarnitines: a population-based study

It is not appropriate for everyone, and understanding who benefits most helps researchers make informed decisions about whether the added complexity and cost are justified