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The question, therefore, arises as to whether the co-administration of a high dose of vitamin E could have inhibited access to the brain for CoQ10, via competition for shared lipoprotein or other carrier types, for example

Vitamin E deficiency with normal serum vitamin E concentrations in children with chronic cholestasis

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10.1016/j.jacc.2018.04.040 6 BehersB

This supplement may decrease the effectiveness of thyroid medications, potentially leading to symptoms of hypothyroidism

Polymerase chain reaction showed that CGA could down-regulate the expression of anti-apoptotic genes Bcl-2 and Bcl-XL , as well as up-regulate the expression of pro-apoptotic genes Bax, Bcl-XS , and Bad (34, 35)
