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Finally, a pervasive sense of being overwhelmed by the cumulative burden of these multisystem symptoms can serve as a valuable clinical indicator for nociplastic pain [12, 75]

doi: 10.1111/acel.13319

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The net result of these actions is inhibition of C-fibre-evoked postsynaptic excitation, although TRPA1-mediated calcium influx may initially increase spontaneous excitatory postsynaptic currents via activation of ionotropic glutamate receptors (iGluR)

Matrix metalloproteinase regulation: These enzymes break down damaged tissue to allow new tissue formation