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In summary, the main approach in the search for new DMDs for AD is the targeting of the pathophysiological processes that cause the onset of the disease, which, according to current AD hypotheses, are A and tau pathology, mitochondrial dysfunction, oxidative stress, neuroinflammation, disturbed neurotransmission, and disorders of brain metabolism

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To first validate the negative control compound, we demonstrated that UNC12567 did not support ternary complex formation between FBXO22 and NSD2 over SEC (Supplementary Fig

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Mitochondrial complex I as a pathologic and therapeutic target for Parkinsons disease

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