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Although intracellular L-arginine concentration typically far exceeds its K m for eNOS, obesity, diabetes, and cardiovascular diseases can cause L-arginine deficiency

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In the LRP6-centric view, as constitutively activated forms of LRP6 fully activate -catenin signaling in an apparently Fz and Dvl-independent manner (He et al., 2004), LRP6 represents the key output whereas Fz and Dvl act upstream to control LRP6 activation

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[DOI] [PMC free article] [PubMed] [Google Scholar] 20.Eleftheriadou A.M., Mehl S., Renko K., Kasim R.H., Schaefer J.A., Minich W.B., Schomburg L

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