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Dimas, P

Before we could continue our investigation, however, we wished to validate a published dopamine depletion model [25]

Therefore, targeting c-MET along with other receptor tyrosine kinases can bring effective therapeutic strategies

where abnormal -synuclein aggregation is visible (Sweeney et al

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Cagrilintide functions primarily through amylin receptor complexes, which are heterodimers formed by the calcitonin receptor and receptor activity modifying proteins
