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The specific mechanism may also be by activating VEGF-A / VEGFR1 -mediated AKT and p38/MAPK signaling pathways to upregulate downstream eNOS expression, which may increase the NO product synthesis, thus promote angiogenesis of gastric mucosa and also interact with endoplasmic reticulum (ER) stress

Epigenetic modifications in the biology of nonalcoholic fatty liver disease the role of DNA Hydroxymethylation and TET proteins

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