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This suggests that enzymes present in the E

NF-B not only induces canonical pro-inflammatory cytokines (TNF-, IL-6, pro-IL-1) but simultaneously up-regulates facilitative glucose transporter, metabolically locking macrophages, DCs and activated T cells into a glycolytic, NO-rich phenotype that fuels chronic mucosal inflammation (11, 110112)

To view a copy of this license, visit About this article Cite this article Trabjerg, M.S., Andersen, D.C., Huntjens, P

Front Med

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