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In alcoholic liver disease, fatty acid synthesis and lipid deposition increases dramatically due to the inhibition of FA oxidation by excess NADH synthesis, the accumulation of acetylCoA (precursor for FA synthesis) as a result of acetate metabolism and the upregulation of fatty acid synthesis machinery through the transcriptional activation of SREBP1c (Osna et al., 2017)

Fernndez M, Javaid F, Chudasama V

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But, then TP53 in the cytoplasm can inhibit protective autophagy

7 These pretreatment doses and times were selected based on other previous studies 1 , 7 and preliminary experiments in which the safety after injection was determined, since we used rats as the experimental animals

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