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in particular, mitochondrial acyl-CoA synthetases are likely to activate fatty acids for transport into the mitochondrial matrix, where the fatty acids are then subject to -oxidation [18,19]

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trans double bonds are very rare naturally (a little bit in some animal fats) but can of course be produced artificially

This is an intriguing finding given that we had observed decreases in ACE2 receptor levels in response to supplementation with LCLT in previous human study (16)