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doi: 10.1185/03007995.2014.909394 6 de VriesMAKlopBEskesSAvan der LoosTLJMKlessens-GodfroyFJMWieboltJet al

Clin Cancer Res 6(1):250-9, 2000

This was not due to decreased bulk acetyl-CoA (Extended Data Fig

The literature reports that carnitine can promote the excretion of excess organic or fatty acids in patients with defects in fatty acid metabolism and/or specific organic acidopathies that bioaccumulate acylCoA esters.1-6 Secondary carnitine deficiency can be a consequence of inborn errors of metabolism or iatrogenic factors such as hemodialysis

Exemplary case from study group 4 (no-treatment control)

Carnitine synthesis defect and autism With the overall goal to identify exonic copy number variants in the genome of ASD patients, Celestino-Soper and coauthors[42] analyzed 3743 samples for detecting disease-causing copy number variants (CNVs) that are not detected by most techniques used in conventional research and clinical diagnosis laboratories