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Recent animal studies have examined how to treat senescent cells with therapies such as the FOXO4-DRI peptide

Kato S, Pinto M, Carvajal A, et al

Garg (New Delhi, India) Objective: To describe the diverse clinical presentations of four cases with the same genotype of abnormality of vitamin B12 metabolism Background: Cobalamin C (CbC) deficiency is a rare autosomal recessive disorder that affects metabolism of methylmalonate and cobalamin, secondary to mutation in the MMACHC gene

Gut microbiota alterations promote traumatic stress susceptibility associated with p-cresol-induced dopaminergic dysfunctions

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This process may ultimately affect the brains interoceptive network, particularly the insular cortex, resulting in altered perception and processing of signals related to fatigue and pain