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The only way to be sure that Se status was low before disease occurs is to measure erythrocyte Se which, with a half-life of 120 d, will only vary after a longer period of inflammation (Reference Stefanowicz, Talwar and O'Reilly46)

Prism 9 (GraphPad Software) was used for statistical analysis of all lifespan, qRT-PCR, and slow-killing assay experiments

doi: 10.1016/j.chemosphere.2019.125492

This causes an uncoordinated release of interferon-gamma and interleukin-2, precipitating sudden and profound hemodynamic collapse

In parallel, studies have reported modulation of early growth response-1 (EGR-1) expression and its regulatory partner NAB2, which are involved in cytokine signaling and early extracellular matrix formation during cellular response processes [2]

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