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Drugdrug interaction profile of the all-oral anti-hepatitis C virus regimen of paritaprevir/ritonavir, ombitasvir and dasabuvir

Myocardial scarring is thought to either induce or maintain ventricular arrhythmias in several ways: 1) scarring creates insulating barriers that form alterations in conduction in the fibrotic tissue, resulting in re-entrant wave fronts of excitation

Because the substrate-binding site does not overlap with this acidic site, it was proposed that protonation of the critical aspartate (D41) by extracellular protons causes TM1 bending, the collapse of the N-lobe cavity and concomitant extrusion of the substrate into the periplasm, and eventually the return to the inward-open state

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