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Although ciclopirox displayed promising results against cystic fibrosis lung infections during in vitro experiments, its potential cytotoxicity should be considered [12]

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Previous research in our laboratory has suggested that physiological concentrations of NSAIDs (diclofenac, naproxen and meclofenamate sodium) cause mitochondrial and proteasome dysfunction in cardiac tissue 12,13

Natural synergistic potential with GLP-1 drugs Because its receptor is completely different from that of GLP-1, it naturally complements GLP-1 receptor agonists such as smegglutinin in terms of mechanism

29 ChenB.ChenZ.LiuM.GaoX.ChengY.WeiY.et al (2019)

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