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The Ecuadorian Laron and Brazilian Itabaianinha cohorts with congenital mutations in the GH and GHRH receptors, respectively, both provide a unique and arguably the purest examples to evaluate the effects of chronic severe IGF-I and both severe GH and IGF-I deficiency, respectively, with severe hypoinsulinemia being the common denominator in both cohorts (151, 152) ( Table 1

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Introduction The growth hormone (GH)-insulin-like growth factor I (IGF-I) axis plays a critical role in promoting linear growth in children ( Insulin, when secreted from pancreatic -cells, is transported by the portal vein directly to the liver resulting in high hepatocyte exposure

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