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S-glutathionylation enhances the fatty acid binding and nuclear accumulation of FABP5, activates PPAR/, and inhibits the LPS-induced inflammation in macrophages

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Drugs acting at synaptic or neuroeffector junctional sites cholinergic agents, cholinergic blocking agents, neuromuscular blocking agents, adrenergic agents, antiadrenergic agents, cholinergic agents as disclosed in U

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The pathophysiology is complex and involves autonomic dysfunction, endocrine disturbances, and reactive mood disorders (i.e., depression or anxiety), combined with genetic, environmental, and socio-economic predispositions [23]