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Arsenic trioxide as an inducer of apoptosis and loss of PML-RAR protein in acute promyelocytic leukemia cells

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Within the liposome-hydrogel system, TPP-CeO 2 mimics the enzymatic activities of SOD and CAT through a reversible Ce 3+ /Ce 4+ redox cycling mechanism

A practical workup combines symptoms, diet, medication history, whole-blood histamine, homocysteine, SAM, SAH, copper/zinc balance, pyrroles and toxic-burden clues

Possible mechanisms could include high levels of non-esterified fatty acids from accelerated lipolysis, oxidative stress propagated by microvascular disease, and pro-inflammatory cytokines [22]