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Consistently, leptin-induced increase in MAP and HR was completely blocked by chronic ICV infusion of MC3/4R antagonist SHU-9119 in rats (Figure 1)
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We found that phosphorylation of the S114 priming residue altered the nucleocytoplasmic distribution of NHR-49 and was necessary for subsequent phosphorylation of adjacent residues by recombinant KIN-19 (Figs
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