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77 , 78 Evidence to support this model stems from a recent study showing that the absence of neuroligin-3 can induce nonsyndromic autism in mice

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this facilitates their gradual release in the intestines, thereby prolonging their antioxidant activity (121)

Additional evidence suggests it plays an important role in immune surveillance and inflammatory cell trafficking, while also allowing for communication between the local tissue environment and the more luminally located smooth muscle and endothelial cells (178, 179)

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