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However, there is no scientific evidence to prove this
Furthermore, many researchers have found that reduced GSH levels and GSH-dependent antioxidant enzyme activity in damaged myocardium due to excess ROS and disruption of the GSH synthesis process, along with increased oxidative damage and ferroptosis
RAAS, renin-angiotensin-aldosterone system
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Ketone body modulation of ligand-gated ion channels
Hydrogen Sulfide Opens the KATP Channel on Rat Atrial and Ventricular Myocytes
