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[DOI] [PMC free article] [PubMed] [Google Scholar] Pizzimenti S., Ciamporcero E., Daga M., Pettazzoni P., Arcaro A., Cetrangolo G., Minelli R., Dianzani C., Lepore A., Gentile F., Barrera G

Am J Physiol Renal Physiol 289: F10401047

, sevoflurane) are well known to cause ischemic preconditioning part of this effect is via mild uncoupling of mitochondria and activation of cytoprotective kinases (like PKC-) that ultimately reduce mPTP opening on reperfusion (128, 129)

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Peptide therapy is generally well-tolerated, with minimal side effects
Current Pharmaceutical Design, 23(27), 4012-4028
