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Our work demonstrates that CHAC1 catabolism of GSH is a significant factor in the depletion of GSH across cellular compartments

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HIF-overexpression and pro-inflammatory priming in human mesenchymal stromal cells improves the healing properties of extracellular vesicles in experimental Crohns disease

Measurements were performed with the same microscope and settings as in our previous study (Horvth et al

Nitrosation-dependent caveolin 1 phosphorylation, ubiquitination, and degradation and its association with idiopathic pulmonary arterial hypertension