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The relationship of increases in these markers of oxidative damage to redox-modulated physiological responses of muscle to exercise is unclear as is the desirability of attempting to suppress these changes through the use of antioxidants

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Therefore, targeting core mechanisms including microglial metabolic reprogramming, key receptor signaling, epigenetic regulation, and toxic phenotype induction to rebalance polarization represents a critical strategy for mitigating neuroinflammation and neurodegeneration in PD