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These alterations enhance the compounds capacity to engage with bacterial targets (Huber 2023)
Additionally, in the presence of PHOXN, a potent RTA, 4-HNE treatment still caused inhibition of LDE-adduct export, identifying LDEs produced following lipid peroxidation as the cause of altered MRP1 function during ferroptosis
~10 minutes for native IGF-1) and reduced binding to the IGF-binding proteins that normally regulate the hormone in the bloodstream
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